Search results for "GABA-A Receptor Antagonists"

showing 10 items of 17 documents

Allopregnanolone augments epileptiform activity of an in-vitro mouse hippocampal preparation in the first postnatal week.

2019

Abstract In the immature brain the neurotransmitter γ-amino butyric acid (GABA) mediates a membrane depolarization and can contribute to both, inhibition and excitation. Therefore the consequences of a positive modulation of GABA(A) receptors by neurosteroids on epileptiform activity are hard to predict. In order to analyze whether neurosteroids attenuate or exaggerate epileptiform activity in the immature brain, we investigated the effect of the neurosteroid allopregnanolone on epileptiform activity in an in-toto hippocampus preparation of early postnatal mice (postnatal days 4–7) using field potential recordings. These in-vitro experiments revealed that 0.5 μmol/L allopregnanolone had no …

0301 basic medicineNeuroactive steroidPatch-Clamp TechniquesPregnanoloneHippocampal formationHippocampusMembrane Potentials03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineAnimalsPicrotoxinIctalGABA-A Receptor AntagonistsNeurotransmitterGABAA receptorAllopregnanoloneDepolarizationnervous system diseases030104 developmental biologynervous systemNeurologychemistryGABAergicNeurology (clinical)Neuroscience030217 neurology & neurosurgeryEpilepsy research
researchProduct

Modulatory effects of the novel TrkB receptor agonist 7,8-dihydroxyflavone on synaptic transmission and intrinsic neuronal excitability in mouse visu…

2013

7,8-Dihydroxyflavone (7,8 DHF) is a new recently identified TrkB receptor agonist, which possesses a potent neurotrophic activity and shares many physiological properties with the neurotrophin "Brain Derived Neurotrophic Factor" (BDNF). However, its precise mechanism of action at the cellular level has not been clarified yet. In the present study we explored the effects of this agent on synaptic and intrinsic neuronal properties by performing whole-cell patch clamp recordings from layer 2/3 pyramidal neurons. Incubation of acute cortical slices with 7,8-DHF (20 µM) for 30 min caused a selective reduction in the strength of GABAergic inhibition. The amplitude of evoked inhibitory postsynapti…

Agonistmedicine.drug_classNerve Tissue ProteinsTropomyosin receptor kinase BNeurotransmissionIn Vitro Techniques78-DihydroxyflavoneInhibitory postsynaptic potentialSynaptic TransmissionGlutamatergicMicemedicineElectric ImpedanceAnimalsReceptor trkBGABA-A Receptor AntagonistsGABAergic NeuronsProtein Kinase InhibitorsCells CulturedNootropic AgentsVisual CortexPharmacologyBrain-derived neurotrophic factorbiologyPyramidal CellsNeural InhibitionFlavonesReceptors GABA-AMice Inbred C57BLKineticsNeuroprotective Agentsbiology.proteinEvoked Potentials VisualNeuroscienceNeurotrophinEuropean journal of pharmacology
researchProduct

Enhanced tonic GABAA inhibition in typical absence epilepsy

2009

The cellular mechanisms underlying typical absence seizures, which characterize various idiopathic generalized epilepsies, are not fully understood, but impaired γ-aminobutyric acid (GABA)-ergic inhibition remains an attractive hypothesis. In contrast, we show here that extrasynaptic GABAA receptor–dependent 'tonic' inhibition is increased in thalamocortical neurons from diverse genetic and pharmacological models of absence seizures. Increased tonic inhibition is due to compromised GABA uptake by the GABA transporter GAT-1 in the genetic models tested, and GAT-1 is crucial in governing seizure genesis. Extrasynaptic GABAA receptors are a requirement for seizures in two of the best character…

GABA Plasma Membrane Transport ProteinsGABA Plasma Membrane Transport ProteinsCellular pathologystargazerBiologyPharmacologytonic currentSettore BIO/09 - FisiologiaArticleGeneral Biochemistry Genetics and Molecular BiologyTonic (physiology)spike–and–wave discharge03 medical and health sciencesEpilepsy0302 clinical medicineThalamusthalamusGenetic modelmedicineAnimalsGABA transporterGABA-A Receptor AntagonistsReceptorTHIP030304 developmental biology0303 health sciencesextrasynaptic tonic current GAT–1 thalamus spike–and–wave discharge GAERS stargazer lethargic GHB THIPGABAA receptorAminobutyratesPetit mal epilepsyGeneral Medicineextrasynapticmedicine.diseaseReceptors GABA-ARats3. Good healthEpilepsy Absenceabsence epilepsy GABA electrophysiology patch clampnervous systemGAT–1GAERSbiology.proteinlethargicGHB030217 neurology & neurosurgery
researchProduct

Drug interactions at GABA(A) receptors.

2002

Neurotransmitter receptor systems have been the focus of intensive pharmacological research for more than 20 years for basic and applied scientific reasons, but only recently has there been a better understanding of their key features. One of these systems includes the type A receptor for the gamma-aminobutyric acid (GABA), which forms an integral anion channel from a pentameric subunit assembly and mediates most of the fast inhibitory neurotransmission in the adult vertebrate central nervous system. Up to now, depending on the definition, 16-19 mammalian subunits have been cloned and localized on different genes. Their assembly into proteins in a poorly defined stoichiometry forms the basi…

GABAA receptorChemistryGeneral NeuroscienceMolecular Sequence DataLoreclezoleNeurotransmissionReceptors GABA-AGABAA-rho receptorGABA AntagonistsNeurotransmitter receptormedicineAnimalsHumansDrug InteractionsAmino Acid SequenceGABA-A Receptor AgonistsGABA-A Receptor AntagonistsBinding siteReceptorGlycine receptorNeuroscienceGABA Agonistsmedicine.drugProgress in neurobiology
researchProduct

Mild systemic inflammation and moderate hypoxia transiently alter neuronal excitability in mouse somatosensory cortex

2016

During the perinatal period, the brain is highly vulnerable to hypoxia and inflammation, which often cause white matter injury and long-term neuronal dysfunction such as motor and cognitive deficits or epileptic seizures. We studied the effects of moderate hypoxia (HYPO), mild systemic inflammation (INFL), or the combination of both (HYPO + INFL) in mouse somatosensory cortex induced during the first postnatal week on network activity and compared it to activity in SHAM control animals. By performing in vitro electrophysiological recordings with multi-electrode arrays from slices prepared directly after injury (P8–10), one week after injury (P13–16), or in young adults (P28–30), we investig…

Male0301 basic medicineAction PotentialsKainate receptorStimulationPotassium ChlorideMicechemistry.chemical_compound0302 clinical medicineHypoxia6-Cyano-7-nitroquinoxaline-23-dioneNeuronsAge FactorsInterleukin-1βElectrophysiologyEpileptiform activityNeurologyAnesthesiaCNQXNMDA receptorFemalemedicine.symptommedicine.drugmedicine.medical_specialtyAMPA receptorIn Vitro TechniquesBiologyBicucullineMulti-electrode arrayArticlelcsh:RC321-57103 medical and health sciencesInternal medicinemedicineAnimalsGABA-A Receptor Antagonistslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryInflammationSystemic inflammationSomatosensory CortexHypoxia (medical)BicucullineBarrel cortexMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinology2-Amino-5-phosphonovalerateGene Expression Regulationchemistrynervous systemExploratory BehaviorExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryNeurobiology of Disease
researchProduct

Opposite motor responses elicited by ethanol in the posterior VTA: The role of acetaldehyde and the non-metabolized fraction of ethanol

2013

Recent electrophysiological evidence suggests that ethanol simultaneously exerts opposite effects on the activity of dopamine (DA) neurons in the ventral tegmental area (VTA) through two parallel mechanisms, one promoting and the other reducing the GABA release onto VTA DA neurons. Here we explore the possible behavioural implications of these findings by investigating the role displayed by acetaldehyde (the main metabolite of ethanol) and the non-metabolized fraction of ethanol in motor activity of rats. We analyse the appearance of motor activation or depression after intra-VTA administration of ethanol in rats subjected to different pharmacological pre-treatments designed to preferential…

MaleMicroinjectionsMetaboliteGABA(A) receptorsAcetaldehydePharmacologyMotor ActivityNon-metabolized fraction of ethanolBicucullineCellular and Molecular Neurosciencechemistry.chemical_compoundDopaminemedicineAnimalsGABA-A Receptor AntagonistsEnzyme InhibitorsRats WistarPharmacologyEthanolDose-Response Relationship DrugEthanolChemistryGABAA receptorVentral Tegmental AreaAcetaldehydeCentral Nervous System DepressantsBicucullineRatsVentral tegmental areaElectrophysiologymedicine.anatomical_structureBiochemistrynervous systemCyanamideVTAmedicine.drug
researchProduct

Effects of desipramine and alprazolam in the forced swim test in rats after long-lasting termination of chronic exposure to picrotoxin and pentylenet…

1993

Abstract Rats were treated for 5 weeks with three subconvulsant doses of picrotoxin (PTX) and pentylenetetrazol (PTZ) per week to induce a persistent reduction of the GABA A receptor function which results in chemical kindling. Fifteen days after termination of this treatment schedule, the effect of desipramine (DMI) and alpraxolam (ALP) on immobility time in the forced swim test (FST) was evaluated. Chronic PTX and PTZ did not alter the immobility time. Acute PTX and PTZ reduced the immobility of rats chronically treated with vehicle but not of those exposed chronically to PTX and PTZ. Chronic PTX did not influence the anti-immobility effect of DMI, but blocked that of ALP. Chronic PTZ mar…

MalePharmacologyMotor ActivityChlordiazepoxidechemistry.chemical_compoundDesipraminemedicineAnimalsPicrotoxinPharmacology (medical)GABA-A Receptor AntagonistsPentylenetetrazolBiological PsychiatrySwimmingPharmacologyAlprazolamGABAA receptorKindlingbusiness.industryDesipramineChlordiazepoxideRatsSubstance Withdrawal SyndromePsychiatry and Mental healthNeurologyAlprazolamchemistryPentylenetetrazoleNeurology (clinical)businesshuman activitiesPsychomotor Performancemedicine.drugBehavioural despair testPicrotoxinEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
researchProduct

Lunasin-induced behavioural effects in mice: Focus on the dopaminergic system

2013

The present study for the first time is devoted to identify central effects of synthetic lunasin, a 43 amino acid peptide. A markedly expressed neuroleptic/cataleptic effect was observed at low (0.1-10 nmol/mouse) centrally administered doses in male C57Bl/6 mice. Lunasin considerably reduced the amphetamine hyperlocomotion but weakly apomorphine climbing behaviour. No influence on ketamine and bicuculline effects was observed. Binding assay studies demonstrated modest affinity of lunasin for the dopamine D₁ receptor (Ki=60 ± 15 μM). In a functional assay of cAMP accumulation on live cells lunasin antagonised apomorphine effect on D₁ receptor activation (pEC₅₀=6.1 ± 0.3), but had no effect …

Malemedicine.medical_specialtyApomorphineDopamine AgentsMotor ActivityPharmacologyBicucullineLunasinBehavioral NeuroscienceDopamine receptor D1SeizuresDopamineInternal medicineCyclic AMPmedicineAnimalsHumansGABA-A Receptor AntagonistsAmphetamineReceptorCatalepsyReceptors Dopamine D2ChemistryReceptors Dopamine D1DopaminergicBrainMice Inbred C57BLApomorphineAmphetamineHEK293 CellsEndocrinologyDopamine receptorSoybean ProteinsKetamineExcitatory Amino Acid AntagonistsCentral Nervous System Agentsmedicine.drugBehavioural Brain Research
researchProduct

Blunted furosemide action on cerebellar GABAA receptors in ANT rats selectively bred for high alcohol sensitivity.

1996

Furosemide specifically reverses the inhibition by gamma-aminobutyric acid (GABA) of t-[35S]-butylbicyclophosphorothionate ([35S]TBPS) binding and increases the basal [35S]TBPS binding to the cerebellar granule cell layer GABAA receptors. For the selectivity of furosemide, an interplay between GABAA receptor alpha 6 and beta 2 or beta 3 subunits is needed. We have now investigated the furosemide sensitivity of cerebellar [35S]TBPS binding in the alcohol-sensitive (ANT) rat line that harbors a pharmacologically critical point mutation in the alpha 6 subunit [alpha 6 (Q1000)], increasing benzodiazepine affinity of the normally insensitive alpha 6-containing receptors. ANT receptors were less …

Malemedicine.medical_specialtyCerebellumPharmacologyLigandsTransfectionGABAA-rho receptorCell LineCellular and Molecular Neurosciencechemistry.chemical_compoundFurosemideInternal medicineCerebellummedicineAnimalsHumansGABA-A Receptor AntagonistsReceptorDiureticsGABA AgonistsIn Situ HybridizationPharmacologyEthanolGABAA receptorAntagonistFurosemideCentral Nervous System DepressantsRats Inbred StrainsReceptors GABA-AANTRecombinant ProteinsRatsEndocrinologymedicine.anatomical_structurenervous systemMuscimolchemistryAutoradiographyFemalemedicine.drugNeuropharmacology
researchProduct

gamma-Aminobutyric acid type A/benzodiazepine receptors regulate rat retina neurosteroidogenesis.

1995

Abstract It has been previously shown that retinal ganglion cells have the ability to synthesize steroids including neuroactive steroids such as pregnenolone sulfate. Since ganglion cells possess GABAA/benzodiazepine (BZ) receptors and neurosteroids modulate retinal GABAA receptor function, we investigated the role of these receptors in isolated rat retina neurosteroidogenesis. Ligands for central-type BZ receptors stimulated retinal pregnenolone synthesis. Clonazepam was the most potent ligand examined acting at nanomolar concentrations. Moreover, the effective steroidogenesis stimulatory dose (ED50) for these ligands and theKi to inhibit [3-H]flunitrazepam binding showed a coefficient of …

Malemedicine.medical_specialtyNeuroactive steroidFlunitrazepamBiologyPharmacologyIn Vitro TechniquesRetinal ganglionSynaptic TransmissionRetinaGABAA-rho receptorchemistry.chemical_compoundInternal medicinemedicineAnimalsGABA-A Receptor AgonistsGABA-A Receptor AntagonistsRats WistarMolecular BiologyNeurotransmitter AgentsGABAA receptorGeneral NeuroscienceBicucullineIsoquinolinesReceptors GABA-ARatsKineticsEndocrinologychemistryMuscimolPregnenolonePregnenoloneSteroidsNeurology (clinical)Pregnenolone sulfateNeurogliaDevelopmental Biologymedicine.drugBrain research
researchProduct